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Disclaimer • Welcome • Why Alternatives? • Alternative Cancer Therapy Guides |
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Introduction and complete list of
Healing Cancer Naturally articles on ”known”, suspected and probable causes of cancer Breast Cancer and Cancer Bacteria ctd. Detecting acid-fast bacteria in breast cancerAs discovered by Virginia Livingston, the acid-fast stain is the essential stain to detect cancer bacteria in histopathologic microscopic tissue sections from
breast cancer. Using acid-fast staining techiques, bacteria have been identified in breast cancer, lymphoma, Kaposi’s sarcoma (the so-called ”gay cancer” of AIDS) and other forms of cancer. [23-25]
Figure 1-5 show bacteria identified in breast cancer and in the metastasis to the skin. Figure 6-8 show the appearance of Staphylococcus epidermidis cultured from the breast tumor metastasis to the skin. All these microphotographs are from a woman who died of breast cancer at age 40, one year after her breast cancer and several positive lymph nodes were removed. A careful perusal of these photographs reveals that the cocci cultured from the tumor are similar, if not identical, to the coccoid forms seen in the original breast cancer tissue. Smaller numbers of microbes were also identified in ”normal” and cancer-free breast tissue removed at the time of surgery. This suggests that the bacteria are not ”secondary invaders” because they are identifiable in areas before the
tissue has been invaded by cancer cells. [23] Click on each figure to see a larger, higher-resolution rendering.
Fig. 1: Histopathologic tissue section from infiltrating ductal carcinoma of the breast. Long arrows point to a cluster of intracellular coccoid forms; short arrows point to scattered extracellular coccoid forms. Intensified Kinyoun’s (acid-fast) stain; magnification x 1000, in oil.
Fig. 2: Tissue section of breast cancer. The long arrow points to a collection of variably-sized round & coccoid forms tightly packed around a cell nucleus. The larger round forms have the appearance of Russell bodies. Short arrows point to tiny coccoid forms resembling the size of ordinary staphylococci at the periphery of the cell. Intensified Kinyoun’s (acid-fast) stain; magnification x1000, in oil.
Fig. 3: Tissue section of breast cancer. Arrows point to a focus of tiny extracellular coccoid forms. Intensified Kinyoun’s (acid-fast) stain; magnification x1000, in oil.
Fig. 4: Tissue section of breast cancer. Arrows point to a small focus o extracellular coccoid forms scattered among the cancerous cells. Intensified Kinyoun’s (acid-fast) stain, magnification x1000, in oil.
Fig. 6: Gram’s-stained smear of Staphylococcus epidermidis cultured from the metastasis of the breast cancer to the skin, illustrated in fig. 5. The bacteria are Gram-variable; Some of the forms stain purple, the typical color of ”Gram-positive” staphylococci. Other cocci stain pink, suggesting poorly-staining, possible cell-wall-deficient forms of staphylococci.
Fig. 7: Same culture as Fig. 6, but showing the appearance of the staphylococci when stained with the Ziehl-Neelson (acid-fast) stain. Note that the size and shape of the staphylococci are identical in size and shape to the small coccoid forms seen in the original breast tumor (Figures 1-4) and in the skin tumor metastasis (Figure 5). Magnification x1000, in oil.
Fig. 8
: Ziehl-Neelson (acid-fast) stain of Staphylococcus epidermidis cultured from a metastatic skin lesion from breast cancer. Note the large dark-stained granules from
which acid-fast (red and pink) thin, sharp ”spicules” emerge. In their 1970 paper [13], Livingston and Alexander-Jackson showed exactly the same type of acid-fast
spicule growth in culture from the urine of a cancer patient (their Figure 12A). (Their research regarding "a specific type of organism cultivated from malignancy" was
presented at the New York Academy of Sciences in November 1969.) Radical treatment and the need for more bacteria researchThe current lack of knowledge about the cause of breast cancer has resulted in the recommendation of some very expensive and death-defying treatments for this horrendous disease. Bone marrow transplants, which carry a 5% death rate, are being proposed as a routine treatment, at a minimal cost of $100,000 per patient. As described in Karen Stabiner’s To Dance with the Devil: The New War on Breast Cancer (1997), the procedure is not pretty. [26] First, a catheter is placed in a woman’s chest to deliver the drugs. A surgical treatment is then performed to scrape out bone marrow from her pelvis, followed by 7 days of growth hormone injections. Then starts days of intravenous chemotherapy that can cause kidney and bladder damage. A catheter is placed in the bladder, followed by a round of intravenous BCNU, or carmustine, a drug that makes a woman feel like she is falling down drunk. Patients become sleepy, sullen, disoriented, agitated, and angry. Loss of bowel control and vomiting are common. After all this, women are put into isolation because the white count drops precipitously, making her vulnerable to all sorts of infections. There may be inexplicable spiking fevers and rashes, and the inevitable loss of hair. After three weeks, patients are allowed to go home where they are told to watch for ”interstitial pneumonitis,” a potentially fatal aftereffect if not diagnosed and treated early. Bone marrow transplant for breast cancer is not guaranteed, nor is it considered a cure. Women have been known to die of cancer three months after the procedure, proving that some patients do not respond to chemotherapy no matter how high the dose. Even with radiation, chemotherapy and surgery, the cost of dying of cancer is not cheap. At the price patients are paying, physicians should not have the luxury of being ignorant about cancer microbe research, particularly when these microbes can be identified in cancer tumors. With 40,000 American women dying annually from breast cancer, it is time medical science reevaluated the parasite of cancer that James Ewing so casually dismissed in 1919. Perhaps if he hadn’t been so adamant about cancer microbe research, his colleagues might have been able to do more to save him when he himself eventually died of bladder cancer. References: 1. Russell W. An address on a characteristic organism of cancer. Br Med J. 1890; 2:1356-1360. Biography:
You can support this site by buying any of these books (or other items) through my Amazon links (US, UK & Canada) and take advantage of Amazon’s (often) low prices.
"This article was originally published by Patient Focus, Inc. The mission of Patient
Focus is to build humanity back into cancer treatment and make it more bearable while a cure for cancer continues to escape us. Patient Focus has a keen interest in
research pertaining to the microbiology of cancer. Also please note that Dr. Cantwell's work regarding cancer-associated bacteria is not related (in his view) to
the research of Hulda Regehr Clark and her theories of cancer causation." Addendum by Dr. Alan Cantwell “For more than a century a small group of researchers, including myself, have implicated bacteria in cancer (see my book, THE CANCER MICROBE, Aries Rising Press). Now it turns out that a common antibiotic -- doxycycline -- can turn off a gene in mice that leads to liver cancer. Let's hope it doesn't take another century for scientists and physicians to follow up on this, and to explain why they keep ignoring cancer-causing bacteria. For more information on "cancer microbes" -- [do an internet search] and type in those exact words.” Antibiotic can 'turn off cancer'adapted from BBC MMIV Scientists have shown that a common antibiotic can turn off cancer cells in mice, offering hope of new treatments for cancer patients. The antibiotic worked by turning off a gene called Myc, which is known to trigger cancer. Mice remained cancer free for as long as they took the drug. When it was stopped they developed liver cancer, the Stanford University team found. Cancer experts said the Nature study held promise for human cancer drugs. Cancer switchThe findings might also apply to cancers of the breast, bowel and prostate, the researchers hope. This is because all of these cancers, as well as liver cancer, begin in cells that line the body called epithelial cells. According to Cancer Research UK, the gene may contribute to as many as one in seven cancer deaths. ”Drugs blocking Myc might be effective cancer treatments in the future.” Dr Elaine Vickers from Cancer Research UK The Stanford scientists studied mice whose liver cells had been altered to carry a modified Myc gene known to cause cancer. Myc controls cell division. Unlike the normal version of the gene, the modified version stayed permanently switched on, meaning cells were constantly dividing and some became cancerous. Feeding the mice the antibiotic doxycyline turned the faulty Myc gene off so cancer growth was blocked. When the researchers stopped the doxycycline the mice developed aggressive liver cancer. Reintroducing doxycycline into their feed not only turned Myc back off, blocking further cancer growth, but it also turned the cancer cells back to normal. Reversing cancerLead researcher Dr Dean Felsher said: "The exciting thing is you can turn cancer cells into something that appears to be normal." But he said even though the cells looked normal, they still had the ability to become cancerous if the antibiotic were to be stopped. This could explain why some cancers come back after people have had chemotherapy, he said. "This is a terrible cancer. Anything that is encouraging in liver cancer may be important," he said. Dr Elaine Vickers, science information officer for Cancer Research UK, said: "The Myc gene is known to be overactive in many types of cancer."Estimates suggest that the gene may contribute to as many as one in seven cancer deaths. "This research is very interesting. "It adds to the weight of evidence suggesting that drugs blocking Myc might be effective cancer treatments in the future." Compare Cancer Causes, On Differences Between Species: Animal Experiment Results Often Not Transferable to Humans, Breast Cancer Treatment Advice & Healing Cancer Naturally.
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